Evidence-based articles on metabolism, insulin resistance, blood sugar, obesity, weight management, and metabolic disease risk. This category explains the science behind metabolic health in practical, cautious language.
8 published articles
GLP 1 medications can produce substantial weight loss, but some lean mass is usually lost along the way. Adequate protein and resistance training may help preserve muscle, although there is no single protein target that fits everyone.
Social media often claims that specific food cravings reveal specific nutrient deficiencies. The biology is more complicated. Here’s what the evidence says about cravings for sugar, salt, chocolate, and carbs.
Artificial and other non-nutritive sweeteners generally do not produce a meaningful acute insulin or glucose spike on their own. They may modestly help with weight control when they replace sugar, but long-term observational studies remain harder to interpret.
Continuous glucose monitors are increasingly marketed to people without diabetes as tools for personalized nutrition and metabolic health. They can reveal glucose patterns and may encourage behavior change, especially in people at higher metabolic risk. But for healthy adults, evidence that CGMs improve long-term health remains limited.
Seed oils such as canola, soybean, corn, and sunflower oil are often described as inflammatory or toxic. But randomized trials and large observational studies do not support many of the strongest claims. The real uncertainties involve food context, repeated high heat cooking, and what these oils replace in the diet.
Walking after a meal can reduce the short-term rise in blood glucose, especially compared with remaining seated. The effect is real, although its long-term importance depends on your overall activity and metabolic health.
Insulin resistance happens when tissues such as muscle, liver, and fat respond less effectively to insulin. Blood sugar may remain normal for years while the pancreas compensates by producing more insulin.
GLP-1 weight-loss drugs can reduce lean mass along with fat. But most of the weight lost is fat, and current evidence does not show that lean-mass loss automatically translates into muscle weakness or pathological muscle wasting.
